Binge eating disorder

Binge eating disorder (BED) is an eating disorder characterized by frequent and recurrent binge eating episodes with associated negative psychological and social problems, but without subsequent purging episodes, such as vomiting.[1]

Binge eating disorder
SpecialtyPsychiatry, clinical psychology
SymptomsEating much faster than normal, eating until feeling uncomfortably full, eating a large amount when not hungry
ComplicationsObesity, tooth decay
CausesUnclear
Risk factorsLow self-esteem, family history of eating disorders, childhood abuse or trauma, anxiety, depression, drug and alcohol use
Diagnostic methodPsychiatry, psychology
Differential diagnosisBulimia nervosa
TreatmentPsychiatry, psychology

BED is a recently described condition,[2] which was required to distinguish binge eating similar to that seen in bulimia nervosa but without characteristic purging. Individuals who are diagnosed with bulimia nervosa and binge eating disorder exhibit similar patterns of compulsive overeating, neurobiological features of dysfunctional cognitive control and food addiction, and biological and environmental risk factors.[3] Some professionals consider BED to be a milder form of bulimia with the two conditions on the same spectrum.[4]

Binge eating is one of the most prevalent eating disorders among adults,[5] though there tends to be less media coverage and research about the disorder in comparison to anorexia nervosa and bulimia nervosa.

Signs and symptoms

Binge eating is the core symptom of BED; however, not everyone who binge eats has BED.[6] An individual may occasionally binge eat without experiencing many of the negative physical, psychological, or social effects of BED. This example may be considered an eating problem (or not), rather than a disorder. Precisely defining binge eating can be problematic,[2] however binge eating episodes in BED are generally described as having the following potential features:

  • Eating much faster than normal,[7] perhaps in a short space of time[8]
  • Eating until feeling uncomfortably full[7]
  • Eating a large amount when not hungry[7]
  • Subjective loss of control over how much or what is eaten[9]
  • Binges may be planned in advance,[7] involving the purchase of special binge foods,[7] and the allocation of specific time for binging, sometimes at night
  • Eating alone or secretly due to embarrassment over the amount of food consumed[7]
  • There may be a dazed mental state during the binge[7]
  • Not being able to remember what was eaten after the binge[7]
  • Feelings of guilt, shame or disgust following a food binge[7][9]

In contrast to bulimia nervosa, binge eating episodes are not regularly followed by activities intended to prevent weight gain,[2] such as self-induced vomiting, laxative or enema misuse, or strenuous exercise.[9] BED is characterized more by overeating than dietary restriction and over concern about body shape.[2] Obesity is common in persons with BED,[2] as are depressive features,[2] low self-esteem, stress and boredom.[8]

Causes

As with other eating disorders, binge eating is an "expressive disorder"—a disorder that is an expression of deeper psychological problems.[3] People who have binge eating disorder have been found to have higher weight bias internalization, which includes low self-esteem, unhealthy eating patterns, and general body dissatisfaction.[10] Binge eating disorder commonly develops as a result or side effect of depression, as it is common for people to turn to comfort foods when they are feeling down.[11]

There was resistance to give binge eating disorder the status of a fully fledged eating disorder because many perceived binge eating disorder to be caused by individual choices.[5] Previous research has focused on the relationship between body image and eating disorders, and concludes that disordered eating might be linked to rigid dieting practices.[12] In the majority of cases of anorexia, extreme and inflexible restriction of dietary intake leads at some point to the development of binge eating, weight regain, bulimia nervosa, or a mixed form of eating disorder not otherwise specified. Binge eating may begin when individuals recover from an adoption of rigid eating habits. When under a strict diet that mimics the effects of starvation, the body may be preparing for a new type of behavior pattern, one that consumes a large amount of food in a relatively short period of time.[13][14][15]

Some studies show that BED aggregates in families and could be genetic. However, very few published studies around the genetics exist.[16]

However, other research suggests that binge eating disorder can also be caused by environmental factors and the impact of traumatic events. One study showed that women with binge eating disorder experienced more adverse life events in the year prior to the onset of the development of the disorder, and that binge eating disorder was positively associated with how frequently negative events occur.[17] Additionally, the research found that individuals who had binge eating disorder were more likely to have experienced physical abuse, perceived risk of physical abuse, stress, and body criticism.[17] Other risk factors may include childhood obesity, critical comments about weight, low self-esteem, depression, and physical or sexual abuse in childhood.[18] A systematic review concluded that bulimia nervosa and binge eating disorder are more impacted by family separations, a loss in their lives and negative parent-child interactions compared to those with anorexia nervosa.[19] A few studies have suggested that there could be a genetic component to binge eating disorder,[5] though other studies have shown more ambiguous results. Studies have shown that binge eating tends to run in families and a twin study by Bulik, Sullivan, and Kendler has shown a, "moderate heritability for binge eating" at 41 percent.[20] More research must be done before any firm conclusions can be drawn regarding the heritability of binge eating disorder. Studies have also shown that eating disorders such as anorexia and bulimia reduce coping abilities, which makes it more likely for those suffering to turn to binge eating as a coping strategy.[21]

A correlation between dietary restraint and the occurrence of binge eating has been shown in some research.[6] While binge eaters are often believed to be lacking in self-control, the root of such behavior might instead be linked to rigid dieting practices. The relationship between strict dieting and binge eating is characterized by a vicious circle. Binge eating is more likely to occur after dieting, and vice versa. Several forms of dieting include delay in eating (e.g., not eating during the day), restriction of overall calorie intake (e.g., setting calorie limit to 1,000 calories per day), and avoidance of certain types of food (e.g., "forbidden" food, such as sugar, carbohydrates, etc.) [22] Strict and extreme dieting differs from ordinary dieting. Some evidence suggests the effectiveness of moderate calorie restriction in decreasing binge eating episodes among overweight individuals with binge eating disorder, at least in the short-term.[23][24]

“In the U.S, it is estimated that 3.5% of young women and 30% to 40% of people who seek weight loss treatments, can be clinically diagnosed with binge eating disorder.”[25]

Diagnosis

International Classification of Diseases

BED was first included in the Diagnostic and Statistical Manual of Mental Disorders (DSM) in 1994 simply as a feature of eating disorder. In 2013 it gained formal recognition as a psychiatric condition in the DSM-5.[26]

The 2017 update to the American version of the ICD-10 includes BED under F50.81.[27] ICD-11 may contain a dedicated entry (6B62), defining BED as frequent, recurrent episodes of binge eating (once a week or more over a period of several months) which are not regularly followed by inappropriate compensatory behaviors aimed at preventing weight gain.[9]

Diagnostic and Statistical Manual

Previously considered a topic for further research exploration, binge eating disorder was included in the Diagnostic and Statistical Manual of Mental Disorders in 2013.[28] Until 2013, binge eating disorder was categorized as an Eating Disorder Not Otherwise Specified, an umbrella category for eating disorders that don't fall under the categories for anorexia nervosa or bulimia nervosa. Because it was not a recognized psychiatric disorder in the DSM-IV until 2013, it has been difficult to obtain insurance reimbursement for treatments.[29] The disorder now has its own category under DSM-5, which outlines the signs and symptoms that must be present to classify a person's behavior as binge eating disorder. Studies have confirmed the high predictive value of these criteria for diagnosing BED.[30]

According to the World Health Organization's ICD-11 classification of BED, the severity of the disorder can be classified as mild (1-3 episodes/week), moderate (4-7 episodes/week), severe (8-13 episodes/week) and extreme (>14 episodes/week).[26]

One study claims that the method for diagnosing BED is for a clinician to conduct a structured interview using the DSM-5 criteria or taking the Eating Disorder Examination.[26] The Structured Clinical Interview takes no more than 75 minutes to complete and has a systematic approach which follows the DSM-5 criteria. The Eating Disorder Examination is a semi-structured interview which identifies the frequency of binges and associated eating disorder features.[26]

Treatment

Counselling and certain medication, such as lisdexamfetamine and selective serotonin reuptake inhibitor (SSRIs), may help.[31] Some recommend a multidisciplinary approach in the treatment of the disorder.[8]

Counselling

Cognitive behavioral therapy (CBT) treatment has been demonstrated as a more effective form of treatment for BED than behavioral weight loss programs. 50 percent of BED individuals achieve complete remission from binge eating[32] and 68-90% will reduce the amount of binge eating episodes they have.[26] CBT has also been shown to be an effective method to address self-image issues and psychiatric comorbidities (e.g., depression) associated with the disorder.[32] The goal of CBT is to interrupt binge-eating behaviour, learn to create a normal eating schedule, change the perception around weight and shape and develop positive attitudes about one's body.[26] Although this treatment is successful in eliminating binge eating episodes, it does not lead to losing any weight.[33] Recent reviews have concluded that psychological interventions such as psychotherapy and behavioral interventions are more effective than pharmacological interventions for the treatment of binge eating disorder.[34] A meta-analysis concluded that psychotherapy based on CBT not only significantly improved binge-eating symptomatology but also reduced a client's BMI significantly at posttreatment and longer than 6 and 12 months after treatment.[35] There is the 12-step Overeaters Anonymous or Food Addicts in Recovery Anonymous. Behavioral weight loss treatment has been proven to be effective as a means to achieve weight loss amongst patients.[36]

Medication

Lisdexamfetamine is a USFDA-approved drug that is used for the treatment of moderate to severe binge eating disorder in adults.[37]

Three other classes of medications are also used in the treatment of binge eating disorder: antidepressants, anticonvulsants, and anti-obesity medications.[38] Antidepressant medications of the selective serotonin reuptake inhibitor (SSRI) have been found to effectively reduce episodes of binge eating and reduce weight.[38] Similarly, anticonvulsant medications such as topiramate and zonisamide may be able to effectively suppress appetite.[38] The long-term effectiveness of medication for binge eating disorder is currently unknown.[34] For BED patients with manic episodes, risperidone is recommended. If BED patients have bipolar depression, lamotrigine is appropriate to use.[39]

Trials of antidepressants, anticonvulsants, and anti-obesity medications suggest that these medications are superior to placebo in reducing binge eating.[40] Medications are not considered the treatment of choice because psychotherapeutic approaches, such as CBT, are more effective than medications for binge eating disorder. A meta-analysis concluded that using medications did not reduce binge-eating episodes and BMI posttreatment at 6–12 months. This indicates a potential possibility of relapse after withdrawal from the medications.[35] Medications also do not increase the effectiveness of psychotherapy, though some patients may benefit from anticonvulsant and anti-obesity medications, such as phentermine/topiramate, for weight loss.[40]

Blocking opioid receptors leads to less food intake. Additionally, bupropion and naltrexone used together may cause weight loss. Combining these alongside psychotherapies like CBT may lead to better outcomes for BED.[41]

Surgery

Bariatric surgery has also been proposed as another approach to treat BED and a recent meta-analysis showed that approximately two-thirds of individuals who seek this type of surgery for weight loss purposes have BED. Bariatric surgery recipients who had BED prior to receiving the surgery tend to have poorer weight-loss outcomes and are more likely to continue to exhibit eating behaviors characteristic of BED.[32]

Lifestyle Interventions

Other treatments for BED include lifestyle interventions like weight training, peer support groups, and investigation of hormonal abnormalities.

Prognosis

Individuals suffering from BED often have a lower overall quality of life and commonly experience social difficulties.[34] Early behavior change is an accurate prediction of remission of symptoms later.[42]

Individuals who have BED commonly have other comorbidities such as major depressive disorder, personality disorder, bipolar disorder, substance abuse, body dysmorphic disorder, kleptomania, irritable bowel syndrome, fibromyalgia, or an anxiety disorder.[32][38] Individuals may also exhibit varying degrees of panic attacks and a history of attempted suicide.[8]

While people of a healthy weight may overeat occasionally, an ongoing habit of consuming large amounts of food in a short period of time may ultimately lead to weight gain and obesity. Bingeing episodes usually include foods that are high in fat, sugar, and/or salt, but low in vitamins and minerals, as these types of foods tend to trigger the greatest chemical and emotional rewards. The main physical health consequences of this type of eating disorder are brought on by the weight gain resulting from calorie-laden bingeing episodes. Mental and emotional consequences of binge eating disorder include social weight stigma and emotional loss of control.[43] Up to 70% of individuals with BED may also be obese,[8] and therefore obesity-associated morbidities such as high blood pressure[8] and coronary artery disease[8] type 2 diabetes mellitus gastrointestinal issues (e.g., gallbladder disease), high cholesterol levels, musculoskeletal problems and obstructive sleep apnea[32][34][44] may also be present.

Epidemiology

General

The prevalence of BED in the general population is approximately 1-3%.[45]

BED cases usually occur between the ages of 12.4 and 24.7, but prevalence rates increase until the age of 40.[46]

Binge eating disorder is the most common eating disorder in adults.[34]

The limited amount of research that has been done on BED shows that rates of binge eating disorder are fairly comparable among men and women.[47] The lifetime prevalence of binge eating disorder has been observed in studies to be 2.0 percent for men and 3.5 percent for women, higher than that of the commonly recognized eating disorders anorexia nervosa and bulimia nervosa.[32] However another systematic literature review found the prevalence average to be about 2.3% in women and about 0.3% in men.[26] Lifetime prevalence rates for BED in women can range anywhere from 1.5 to 6 times higher than in men.[46] One literature review found that point prevalence rates for BED vary from 0.1 percent to 24.1 percent depending on the sample.[46] This same review also found that the 12-month prevalence rates vary between 0.1 percent to 8.8 percent.[46]

Recent studies found that eating disorders which included anorexia nervosa, bulimia nervosa and binge-eating disorder are common among sexual and gender minority populations, including gay, lesbian, bisexual and transgender people. This could be due to the minority stress and discrimination this population experiences.[48]

Due to limited and inconsistent information and research on ethnic and racial differences, prevalence rates are hard to determine for BED.[46] Rates of binge eating disorder have been found to be similar among black women, white women, and white men,[49] while some studies have shown that binge eating disorder is more common among black women than among white women.[5] However, majority of the research done around BED is focused on White women.[50] One literature review found information citing no difference between BED prevalence among Hispanic, African American, and White women while other information found that BED prevalence was highest among Hispanics followed by Black individuals and finally White people.[46]

Worldwide Prevalences

Eating disorders have usually been considered something that was specific to Western countries. However, the prevalence of eating disorders is increasing in other non-Western countries.[51] Though the research on binge eating disorders tends to be concentrated in North America, the disorder occurs across cultures.[52] In the USA, BED is present in 0.8% of male adults and 1.6% of female adults in a given year.[28]

The prevalence of BED is lower in Nordic countries compared to Europe in a study that included Finland, Sweden, Norway, and Iceland.[51] The point prevalence ranged from 0.4 to 1.5 percent and the lifetime prevalence ranged from 0.7 to 5.8 percent for BED in women.[51]

In a study that included Argentina, Brazil, Chile, Colombia, Mexico, and Venezuela, the point prevalence for BED was 3.53 percent.[53] Therefore, this particular study found that the prevalence for BED is higher in these Latin American countries compared to Western countries.[53]

The prevalence of BED in Europe ranges from <1 to 4 percent.[54]

Co-morbidities

BED is co-morbid with diabetes, hypertension, previous stroke, and heart disease in some individuals.[46]

In people who have obsessive-compulsive disorder or bipolar I or II disorders, BED lifetime prevalence was found to be higher.[46]

Additionally, 30 to 40 percent of individuals seeking treatment for weight-loss can be diagnosed with binge eating disorder.[32]

Underreporting in Men

Eating disorders are oftentimes underreported in men.[51] Underreporting could be a result of measurement bias due to how eating disorders are defined.[51] The current definition for eating disorders focuses on thinness.[51] However, eating disorders in men tend to center on muscularity and would therefore warrant a need for a different measurement definition.[51] Further research should focus on including more men in samples since previous research has focused primarily on women.[51]

History

The disorder was first described in 1959 by psychiatrist and researcher Albert Stunkard as "night eating syndrome" (NES).[55] The term "binge eating" was coined to describe the same bingeing-type eating behavior but without the exclusive nocturnal component.[56]

There is generally less research on binge eating disorder in comparison to anorexia nervosa and bulimia nervosa.[5]

See also

References

  1. Agüera, Zaida; Lozano-Madrid, María; Mallorquí-Bagué, Núria; Jiménez-Murcia, Susana; Menchón, José M.; Fernández-Aranda, Fernando (28 April 2020). "A review of binge eating disorder and obesity". neuropsychiatrie. doi:10.1007/s40211-020-00346-w. ISSN 0948-6259.
  2. Eating disorders: core interventions in the treatment and management of anorexia nervosa, bulimia nervosa, and related eating disorders; National Clinical Practice Guideline No. CG9. Leicester [u.a.]: The British Psychological Society and Gaskell. 2004. ISBN 978-1854333988.
  3. Wu M, Brockmeyer T, Hartmann M, Skunde M, Herzog W, Friederich HC (December 2014). "Set-shifting ability across the spectrum of eating disorders and in overweight and obesity: a systematic review and meta-analysis". Psychological Medicine. 44 (16): 3365–85. doi:10.1017/S0033291714000294. PMID 25066267.
  4. Hay PP, Bacaltchuk J, Stefano S, Kashyap P (October 2009). "Psychological treatments for bulimia nervosa and binging". The Cochrane Database of Systematic Reviews (4): CD000562. doi:10.1002/14651858.CD000562.pub3. PMC 7034415. PMID 19821271.
  5. Saguy A, Gruys K. "Morality and Health: News Media Constructions of Overweight and Eating Disorders" (PDF). UCLA. Retrieved 23 November 2014.
  6. Fairburn C (2013). Overcoming binge eating: the proven program to learn why you binge and how you can stop (Second ed.). New York: Guilford Publications. ISBN 978-1572305618.
  7. "Binge eating disorder - NHS Choices". www.nhs.uk. Nation Health Service. Retrieved 19 January 2017.
  8. Michalska A, Szejko N, Jakubczyk A, Wojnar M (2016). "Nonspecific eating disorders - a subjective review" (PDF). Psychiatria Polska. 50 (3): 497–507. doi:10.12740/PP/59217. PMID 27556109.
  9. "ICD-11 Beta Draft - Mortality and Morbidity Statistics". apps.who.int. Retrieved 19 January 2017.
  10. Pearl RL, White MA, Grilo CM (April 2014). "Overvaluation of shape and weight as a mediator between self-esteem and weight bias internalization among patients with binge eating disorder". Eating Behaviors. 15 (2): 259–61. doi:10.1016/j.eatbeh.2014.03.005. PMC 4053161. PMID 24854815.
  11. "USATODAY.com - Women like sugar, men like meat". usatoday30.usatoday.com. Retrieved 21 April 2016.
  12. Herbozo S, Schaefer LM, Thompson JK (April 2015). "A comparison of eating disorder psychopathology, appearance satisfaction, and self-esteem in overweight and obese women with and without binge eating". Eating Behaviors. 17: 86–9. doi:10.1016/j.eatbeh.2015.01.007. PMID 25668799.
  13. "Binge Eating Disorder". HelpGuide. Retrieved 10 May 2020.
  14. "Where next after anorexia: death, recovery, or another eating disorder?". Psychology Today. Retrieved 21 April 2016.
  15. Veenstra EM, de Jong PJ (August 2010). "Restrained eaters show enhanced automatic approach tendencies towards food". Appetite. 55 (1): 30–6. doi:10.1016/j.appet.2010.03.007. PMID 20298730.
  16. "Genetics of Eating Disorders: What the Clinician Needs to Know". Psychiatric Clinics of North America. 42 (1): 59–73. 1 March 2019. doi:10.1016/j.psc.2018.10.007. ISSN 0193-953X.
  17. Mazzeo SE, Bulik CM (January 2009). "Environmental and genetic risk factors for eating disorders: what the clinician needs to know". Child and Adolescent Psychiatric Clinics of North America. 18 (1): 67–82. doi:10.1016/j.chc.2008.07.003. PMC 2719561. PMID 19014858.
  18. Rayworth BB, Wise LA, Harlow BL (May 2004). "Childhood abuse and risk of eating disorders in women". Epidemiology. 15 (3): 271–8. doi:10.1097/01.ede.0000120047.07140.9d. JSTOR 20485891. PMID 15097006.
  19. Grogan, Katie; MacGarry, Diarmuid; Bramham, Jessica; Scriven, Mary; Maher, Caroline; Fitzgerald, Amanda (December 2020). "Family-related non-abuse adverse life experiences occurring for adults diagnosed with eating disorders: a systematic review". Journal of Eating Disorders. 8 (1): 36. doi:10.1186/s40337-020-00311-6. ISSN 2050-2974. PMC 7374817. PMID 32704372.
  20. Bulik CM, Sullivan PF, Kendler KS (April 2003). "Genetic and environmental contributions to obesity and binge eating". The International Journal of Eating Disorders. 33 (3): 293–8. doi:10.1002/eat.10140. PMID 12655626.
  21. Troop NA, Holbrey A, Treasure JL (September 1998). "Stress, coping, and crisis support in eating disorders". The International Journal of Eating Disorders. 24 (2): 157–66. doi:10.1002/(SICI)1098-108X(199809)24:2<157::AID-EAT5>3.0.CO;2-D. PMID 9697014. Retrieved 21 April 2016.
  22. Tuschl RJ (April 1990). "From dietary restraint to binge eating: some theoretical considerations". Appetite. 14 (2): 105–9. doi:10.1016/0195-6663(90)90004-R. PMID 2186700.
  23. Wilson, Terence (2002). "The controversy over dieting". In Fairburn C, Brownell K (eds.). Eating disorders and obesity: a comprehensive handbook (2nd ed.). New York: Guilford. ISBN 978-1593852368.
  24. Wilfley, Denise (2002). "Psychological treatment of binge eating disorder". In Fairburn C, Brownell K (eds.). Eating disorders and obesity: a comprehensive handbook (2nd ed.). New York: Guilford. ISBN 978-1593852368.
  25. "Eating Disorder Statistics".
  26. Chevinsky, Jonathan D.; Wadden, Thomas A.; Chao, Ariana M. (14 April 2020). "Binge Eating Disorder in Patients with Type 2 Diabetes: Diagnostic and Management Challenges". Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy. doi:10.2147/dmso.s213379. PMC 7166070. PMID 32341661. Retrieved 26 October 2020.
  27. "2017 ICD-10-CM Diagnosis Code F50.81 : Binge eating disorder". www.icd10data.com. Retrieved 8 May 2017.
  28. American Psychiatry Association (2013). Diagnostic and Statistical Manual of Mental Disorders (5th ed.). Arlington: American Psychiatric Publishing. pp. 329–354. ISBN 978-0890425558.
  29. 5 tips: overcome your tendency to overeat . Paper Boys Club, 2 December 2017.
  30. "A Guide to DSM-5: Binge Eating Disorder". Medscape.com. Retrieved 8 June 2013.
  31. Brownley KA, Berkman ND, Peat CM, Lohr KN, Cullen KE, Bann CM, Bulik CM (September 2016). "Binge-Eating Disorder in Adults: A Systematic Review and Meta-analysis". Annals of Internal Medicine. 165 (6): 409–20. doi:10.7326/M15-2455. PMC 5637727. PMID 27367316.
  32. Westerburg DP, Waitz M (November–December 2013). "Binge-eating disorder". Osteopathic Family Physician. 5 (6): 230–33. doi:10.1016/j.osfp.2013.06.003.
  33. Citrome, Leslie (August 2019). "Binge eating disorder revisited: what's new, what's different, what's next". CNS Spectrums. 24 (S1): 4–13. doi:10.1017/S1092852919001032. ISSN 1092-8529.
  34. Iacovino JM, Gredysa DM, Altman M, Wilfley DE (August 2012). "Psychological treatments for binge eating disorder". Current Psychiatry Reports. 14 (4): 432–46. doi:10.1007/s11920-012-0277-8. PMC 3433807. PMID 22707016.
  35. Hilbert, Anja; Petroff, David; Herpertz, Stephan; Pietrowsky, Reinhard; Tuschen‐Caffier, Brunna; Vocks, Silja; Schmidt, Ricarda (September 2020). "Meta‐analysis on the long‐term effectiveness of psychological and medical treatments for binge‐eating disorder". International Journal of Eating Disorders. 53 (9): 1353–1376. doi:10.1002/eat.23297. ISSN 0276-3478.
  36. Wilson GT, Wilfley DE, Agras WS, Bryson SW (January 2010). "Psychological treatments of binge eating disorder". Archives of General Psychiatry. 67 (1): 94–101. doi:10.1001/archgenpsychiatry.2009.170. PMC 3757519. PMID 20048227.
  37. "Vyvanse Prescribing Information" (PDF). United States Food and Drug Administration. Shire US Inc. May 2017. pp. 1–3. Retrieved 10 July 2017.
  38. Marazziti D, Corsi M, Baroni S, Consoli G, Catena-Dell'Osso M (December 2012). "Latest advancements in the pharmacological treatment of binge eating disorder" (PDF). European Review for Medical and Pharmacological Sciences. 16 (15): 2102–7. PMID 23280026.
  39. Himmerich, Hubertus; Kan, Carol; Au, Katie; Treasure, Janet (25 August 2020). "Pharmacological treatment of eating disorders, comorbid mental health problems, malnutrition and physical health consequences". Pharmacology & Therapeutics: 107667. doi:10.1016/j.pharmthera.2020.107667. ISSN 0163-7258.
  40. Lindsay Bodell, Michael Devlin (2011). "Pharmacotherapy for binge-eating disorder". In Grilo C, Mitchell J (eds.). The treatment of eating disorders: a clinical handbook. New York: Guilford. ISBN 978-1609184957.
  41. Valbrun, Leon P.; Zvonarev, Valeriy (2020). "The Opioid System and Food Intake: Use of Opiate Antagonists in Treatment of Binge Eating Disorder and Abnormal Eating Behavior". Journal of Clinical Medicine Research. 12 (2): 41–63. doi:10.14740/jocmr4066. ISSN 1918-3003. PMC 7011935. PMID 32095174.
  42. Nazar BP, Gregor LK, Albano G, Marchica A, Coco GL, Cardi V, Treasure J (March 2017). "Early Response to treatment in Eating Disorders: A Systematic Review and a Diagnostic Test Accuracy Meta-Analysis". European Eating Disorders Review. 25 (2): 67–79. doi:10.1002/erv.2495. PMID 27928853.
  43. "Binge Eating Disorder". National Eating Disorders Association. 26 February 2017. Retrieved 18 February 2019.
  44. "Binge Eating Disorder". National Eating Disorders Association. Retrieved 18 April 2014.
  45. Perkins SJ, Murphy R, Schmidt U, Williams C (July 2006). "Self-help and guided self-help for eating disorders". The Cochrane Database of Systematic Reviews (3): CD004191. doi:10.1002/14651858.CD004191.pub2. PMID 16856036.
  46. Ágh, Tamás; Kovács, Gábor; Pawaskar, Manjiri; Supina, Dylan; Inotai, András; Vokó, Zoltán (March 2015). "Epidemiology, health-related quality of life and economic burden of binge eating disorder: a systematic literature review". Eating and weight disorders: EWD. 20 (1): 1–12. doi:10.1007/s40519-014-0173-9. ISSN 1590-1262. PMC 4349998. PMID 25571885.
  47. Striegel-Moore RH, Rosselli F, Perrin N, DeBar L, Wilson GT, May A, Kraemer HC (July 2009). "Gender difference in the prevalence of eating disorder symptoms". The International Journal of Eating Disorders. 42 (5): 471–4. doi:10.1002/eat.20625. PMC 2696560. PMID 19107833.
  48. Nagata, Jason M.; Ganson, Kyle T.; Austin, S. Bryn (November 2020). "Emerging trends in eating disorders among sexual and gender minorities". Current Opinion in Psychiatry. 33 (6): 562–567. doi:10.1097/YCO.0000000000000645. ISSN 0951-7367.
  49. Mitchison D, Mond J, Slewa-Younan S, Hay P (May 2013). "Sex differences in health-related quality of life impairment associated with eating disorder features: a general population study". The International Journal of Eating Disorders. 46 (4): 375–80. doi:10.1002/eat.22097. PMID 23355018.
  50. Goode, Rachel W.; Cowell, Mariah M.; Mazzeo, Suzanne E.; Cooper‐Lewter, Courtney; Forte, Alexandria; Olayia, Oona‐Ifé; Bulik, Cynthia M. (April 2020). "Binge eating and binge‐eating disorder in Black women: A systematic review". International Journal of Eating Disorders. 53 (4): 491–507. doi:10.1002/eat.23217. ISSN 0276-3478.
  51. Dahlgren, Camilla Lindvall; Stedal, Kristin; Wisting, Line (3 July 2018). "A systematic review of eating disorder prevalence in the Nordic countries: 1994–2016". Nordic Psychology. 70 (3): 209–227. doi:10.1080/19012276.2017.1410071. ISSN 1901-2276.
  52. Pike KM, Hoek HW, Dunne PE (November 2014). "Cultural trends and eating disorders". Current Opinion in Psychiatry. 27 (6): 436–42. doi:10.1097/YCO.0000000000000100. PMID 25211499.
  53. Kolar, David R.; Rodriguez, Dania L. Mejía; Chams, Moises Mebarak; Hoek, Hans W. (November 2016). "Epidemiology of eating disorders in Latin America: a systematic review and meta-analysis". Current Opinion in Psychiatry. 29 (6): 363–371. doi:10.1097/YCO.0000000000000279. ISSN 0951-7367.
  54. Keski-Rahkonen, Anna; Mustelin, Linda (November 2016). "Epidemiology of eating disorders in Europe: prevalence, incidence, comorbidity, course, consequences, and risk factors". Current Opinion in Psychiatry. 29 (6): 340–345. doi:10.1097/YCO.0000000000000278. ISSN 0951-7367.
  55. Stunkard AJ (April 1959). "Eating patterns and obesity". The Psychiatric Quarterly. 33 (2): 284–95. doi:10.1007/BF01575455. PMID 13835451.
  56. Brewerton T. "Binge Eating: Recognition, Diagnosis, and Treatment". Medscape Health eJournal. Retrieved 15 December 2014.

Bibliography

Classification
External resources
This article is issued from Wikipedia. The text is licensed under Creative Commons - Attribution - Sharealike. Additional terms may apply for the media files.