Viral cardiomyopathy

Viral cardiomyopathy occurs when viral infections cause myocarditis with a resulting thickening of the myocardium and dilation of the ventricles. These viruses include Coxsackie B and adenovirus, echoviruses, influenza H1N1, Epstein-Barr virus, rubella (German measles virus), varicella (chickenpox virus), mumps, measles, parvoviruses, yellow fever, dengue fever, polio, rabies and the viruses that cause hepatitis A and C as well as COVID-19 where it has been seen to cause this in persons otherwise thought to be "low risk" of the virus's effects.[1][2][3]

Viral cardiomyopathy
SpecialtyCardiology

See also

References
  1. Barbandi M, Cordero-Reyes A, Orrego CM, Torre-Amione G, Seethamraju H (Jan 2012). "A case series of reversible acute cardiomyopathy associated with H1N1 influenza infection". Methodist DeBakey Cardiovascular Journal. 8 (1): 42–5. doi:10.14797/mdcj-8-1-42. PMC 3405785. PMID 22891110.
  2. Badorff C; Lee G. H.; Knowlton K. U. (2000). "Enteroviral cardiomyopathy: bad news for the dystrophin-glycoprotein complex". Herz. 25 (3): 227–32. doi:10.1007/s000590050011. PMID 10904843. S2CID 25973717.
  3. Mutlu H, Alam M, Ozbilgin OF (2011). "A rare case of Epstein-Barr virus-induced dilated cardiomyopathy". Heart Lung. 40 (1): 81–7. doi:10.1016/j.hrtlng.2009.12.012. PMID 20561866.
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